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In the electron transport chain (ETC), FMN and FAD function as cofactors that shuttle electrons, acting especially in I and II complexes, which contain flavoproteins reductases and electron transferring flavoproteins, thereby facilitating the production of adenosine triphosphate (ATP), the primary energy source of cells, being particularly important in the nervous system, due to its high energy demands (3, 43, 45)
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In fact, it has been shown that UDCA pre-treatment protects cultured rat hepatocytes against oxidative stress induced by ROS, significantly increasing the total amount of GSH and thiol-containing proteins, via the up-regulation of GCL mRNA expression and metallothionein [187]
