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They found that the likelihood of heteroplasmic mutations in non-polymorphic sites, the sites that may produce OXPHOS abnormalities, is 53% higher in ASD children than in unaffected siblings

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“Reversal of Refractory Ulcerative Colitis and Severe Chronic Fatigue Syndrome Symptoms Arising from Immune Disturbance in an HLADR/DQ Genetically Susceptible Individual with Multiple Biotoxin Exposures.” The American Journal of Case Reports, vol

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