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Cytoplasmic FABPs allow schistosomes to acquire fatty acids and cholesterol from the host due to a lack of schistosome oxygen-dependent synthetic pathways essential for membrane formation, protein anchoring, maturation, and egg production (75–79)

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However, in OA, chronic inflammation and oxidative stress often dysregulate lipid metabolism, leading to increased synthesis and accumulation of n-6 PUFAs such as arachidonic acid and linoleic acid